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Therapeutic potential of Janus kinase inhibitors for the management of ...

2. JAK-STAT pathway in intestinal fibrosis 2.1. Key players in intestinal fibrosis Before discussing the potential of JAK inhibitors to treat intestinal fibrosis , it is crucial to understand the underlying pathogenesis. Intestinal fibrosis is characterized by excessive accumulation of ECM together with increased smooth muscle layer thickness.

The Janus kinase-signal transducer and activator of transcription ( JAK-STAT ) pathway plays a crucial role in intestinal homeostasis and inflammation . JAK inhibition represents a relatively novel therapeutic strategy in IBD by simultaneously blocking multiple cytokines across various inflammatory pathways.

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Summary/Conclusion: In an aging-associated MF-like murine model without driver mutations, NF-κB inhibitors , either alone or in combination with JAK inhibitors , as well as dual-acting inhibitors , reduced inflammatory cytokines and splenomegaly, reversed thrombocytopenia and improved BM fibrosis .

Targeting aberrant IFN-γ-associated activity, particularly those linked to inflammatory cascades and abnormal immune system stimulation, could potentially facilitate liver regeneration processes while concurrently inhibiting the progression of fibrosis , thereby enhancing the overall efficacy of liver repair.

Dual Inhibition of NF

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